Endotoxic shock after transhepatic cholangiography.
نویسندگان
چکیده
منابع مشابه
Fatal endotoxic shock of biliary tract origin complicating transhepatic cholangiography.
A 70-year-old man was admitted to hospital with a five-month history of painless jaundice. Previous history included quiescent Pott's disease of the spine, endogenous depression, and Parkinsonism. Exammation showed obvious jaundice and a pronounced lumbar kyphosis. There was no ascites, hepatosplenomegly, or a palpable abdominal tumour. Haematological investigations were normal. The E.S.R. (Wes...
متن کاملPercutaneous transhepatic cholangiography.
Chemical and histological studies are often unable to differentiate chronic extrahepatic obstructive jaundice (due to calculi, neoplasia, or stricture) from chronic intrahepatic obstructive jaundice (caused by primary biliary cirrhosis or by drugs, notably anabolic steroids and phenothiazine derivatives). Oral and intravenous cholangiography are also ineffective when the serum bilirubin is grea...
متن کاملBacteraemic and endotoxic shock.
In 1924 Sanarelli recorded that endotoxins of Gram-negative organisms would damage capillary and postcapillary venular endothelium. By 1935 it was known that two spaced doses of endotoxin given to rabbits would cause renal cortical necrosis and foci of damage in many organs, the Shwartzman reaction. This is due to a combination of vasospasm and intravascular coagulation. Human equivalents of th...
متن کاملNew Methods for Diagnosis and Research PERCUTANEOUS TRANSHEPATIC CHOLANGIOGRAPHY
This is an evaluation of percutaneous transhepatic cholangiography, a technique of over eight years' standing, previously considered by many authorities as both unrewarding and dangerous. The value and risks of the procedure have been examined in 20 patients with obstructive jaundice of uncertain origin and in one further patient with a post-cholecystectomy syndrome. The presence and the nature...
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The inflammatory response induced by bacterial lipopolysaccharide (LPS) has profound metabolic and physiological effects. Thus hepatic glucose production is depressed after LPS administration, which is, at least in part, due to the downregulation of phosphoenolpyruvate carboxykinase (PEPCK) expression. PEPCK is a key regulatory enzyme of the gluconeogenic pathway. Expression of heat shock prote...
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ژورنال
عنوان ژورنال: BMJ
سال: 1973
ISSN: 0959-8138,1468-5833
DOI: 10.1136/bmj.3.5879.541-b